About this item:

86 Views | 23 Downloads

Author Notes:

Shi-Yong Sun, ssun@emory.edu

Authors have no conflicts of interest to declare.

Subject:

Keywords:

  • MEK/ERK
  • acquired resistance
  • lung cancer
  • osimertinib

Re-enforcing the strategy of targeting MEK/ERK signaling to overcome acquired resistance to third generation EGFR inhibitors.

Tools:

Journal Title:

Oncoscience

Volume:

Volume 8

Publisher:

, Pages 80-81

Type of Work:

Article | Final Publisher PDF

Abstract:

Lung cancer, which consists of over 80% non-small cell lung cancer (NSCLC), is the leading cause of cancer-related death in the world [1]. The discovery of the connection between mutant epidermal growth factor receptor (EGFR) and cancer cell response to EGFR-tyrosine kinase inhibitors (EGFR-TKIs) that led to development of EGFR-targeted therapy has revolutionized the treatment of NSCLC. During the last two decades, EGFR-TKIs have advanced from 1st and 2nd generation to 3rd generation agents represented by osimertinib (AZD9291). Because of its promising performance in clinical trials, osimertinib has been approved to treat NSCLC patients with EGFR activating mutations (e.g., exon 19 deletion and exon 21 point mutation L858R) as a frontline therapy or those progressed from 1st or 2nd generation EGFR-TKIs due to EGFR T790M mutation as a second-line treatment option [2, 3].

Copyright information:

© 2021 Zhao and Sun.

This is an Open Access work distributed under the terms of the Creative Commons Attribution 3.0 Unported License (https://creativecommons.org/licenses/by/3.0/rdf).
Export to EndNote