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Author Notes:

Corresponding author: Machelle T. Pardue, Rehabilitation Research and Development Center, Atlanta VA Medical Center, 1670 Clairmont Road, Decatur, GA 30033; mpardue@emory.edu.

The authors thank Tamara Barnett, Paulette Allard, and Kathy Knill for technical assistance and Alison Ziesel for in silico confirmation of RT-PCR primer specificities and complementary locations.

Disclosure: M.J. Phillips, None; S. Webb-Wood, None; A.E. Faulkner, None; S.B. Jabbar, None; V. Biousse, None; N.J. Newman, None; V.T. Do, None; J.H. Boatright, None; D.C. Wallace, None; M.T. Pardue, None

Subject:

Research Funding:

Supported by the Rehabilitative Research and Development Service of the Department of Veterans Affairs, Research to Prevent Blindness, and National Institutes of Health Grants P30 EY06360, P30 AT000609, R01 EY014026, R01-NS41850 (DCW), and R01-NS21328 (DCW).

Retinal Function and Structure in Ant1-Deficient Mice

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Journal Title:

Investigative Ophthalmology & Visual Science

Volume:

Volume 51, Number 12

Publisher:

, Pages 6744-6752

Type of Work:

Article | Final Publisher PDF

Abstract:

Because the retina has high energy demands, the loss of a mitochondrial ATP transporter was predicted to impact retinal function. The authors examined retinal function and morphology in mice without ANT1, a key ATP transporter associated with myopathy.

Copyright information:

© Association for Research in Vision and Ophthalmology

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